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Transient erythema, pruritus, or stinging may develop at the application site, typically resolving with continued use or brief treatment interruption

37 Repressing the various related biomolecular processes involved in inflammation, neurotrophin release and remodeling of ECM components in the IVD might desensitize the intradiscal nociceptive fibers and minimize the acute and/or chronic pain

More importantly, stimulation of insulin secretion and suppression of glucagon by exenatides is strictly dependent on plasma glucose concentrations, thereby minimizing the adverse effect of hypoglycemia

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