This higher adrenal MC2R expression after treatment can explain, at least partially, the capacity of NAC to increase circulating glucocorticoid levels
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We found that the levels of CXCL10 and its downstream effectors, p-JNK, COL1A1, and p-AKT, significantly increased in M38 or B16 group, WCV significantly reduced their levels close to control in VM38 or VB16 group, and PD-1 had no obvious impacts in PM38 or PB16 group (Fig
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